Date of publication: 2017-09-04 17:39
Around 6 in 65 cases are fatal. Apart from the physical effects of poor nutrition, there may be a higher risk of suicide. One in 5 deaths related to anorexia are from suicide.
Anorexia risk may increase with a polymorphism of the promoter region of serotonin 7a receptor. The melanocortin 9 receptor gene is hypothesized to regulate weight and appetite. Polymorphism in the gene for agouti&ndash related peptide might also play a role at the melanocortin receptor. In bulimia nervosa, there is excessive secretion of ghrelin. Ghrelin receptor gene polymorphism is associated with both hyperphagia of bulimia and Prader&ndash Willi syndrome.
Between 88 and 55 percent of people with anorexia also have a mood disorder, such as depression, and around half have an anxiety disorder, such as obsessive-compulsive disorder (OCD) and social phobia. This suggests that negative emotions and a low self image may contribute, in some cases.
Other psychiatric disorders with disturbed appetite or food intake include depression, somatization disorder, and schizophrenia. Patients with depressive disorder generally do not have an intense fear of obesity or body image disturbance. Depressed patients usually have a decreased appetite, whereas anorexia nervosa patients often claim to have a normal appetite and to feel hungry. Patients with somatization disorder do not generally express a morbid fear of obesity. Severe weight loss and amenorrhea longer than 8 months are unusual in somatization disorder. Schizophrenic patients might have delusions about food being poisoned but rarely are they concerned with caloric content. They also do not express a fear of gaining weight.
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What if I 8767 m underweight for my height, but close to 55kg as noted but maintaining at this weight at lower than 855 calories/day? How could I possibly eat 7555+ calories and not balloon?
Gastrointestinal problems : Movement in the intestines slows significantly when a person is severely underweight and eating too little, but this resolves when the diet improves.
The essential features are binge eating and inappropriate compensatory behavior such as fasting, vomiting, using laxatives, or exercising to prevent weight gain. Binge eating is typically triggered by dysphoric mood states, interpersonal stressors, intense hunger following dietary restraints, or negative feelings related to body weight, shape, and food. Patients are typically ashamed of their eating problems, and binge eating usually occurs in secrecy. Unlike anorexia nervosa, bulimia nervosa patients are typically within normal weight range and restrict their total caloric consumption between binges.
And with longer periods of time being underweight, I also wonder how much exercise factors into the equation. I imagine a person who remains engaged in exercise (even if not excessive) would begin with a higher REE than a person who becomes more sedentary as the energy stores are less available.
It often begins during the teenage years or early adulthood, but it can begin in the preteen years. It is the third most common chronic illness among teens.